CV Inflammation Labs

So we could not get the custom live feed
to work yet again we had to just abort that lead that custom live feed so I’m
gonna go ahead and get started here under the well yeah we’re getting some
people joining us. Great. If you could make a couple of comments I would
appreciate it. Thank you very much for your patience. I promise we’re
going to get this technology down sooner or later. We put several more man-hours,
about four more man-hours of client clients time, Michele’s time and about
three more of my time today to try to get us up and rolling. Guess what. It
didn’t work so I say. Loretta Dillon-Ham thank you for your patience Loretta
thanks for joining us. Trump Tribe 2020 I haven’t seen you there before thank you
for joining us thank you for your work. Loretta Dillon-Ham can see you and hear
you you’re doing fine great thank you. David Ivers
good to see you again check again. Chalice of Immortality what’s the
primary best way to prevent the chance of having a heart attack. Well that’s
what all this channel is about. I will say this. I mean, it’s like many
other things. It can get very very complicated but you can simplify and by
saying, first, let’s assess the situation and that was one of the things we were
going to talk about tonight. Here’s what here’s what happens in the typical cardiovascular “prevention” world.
Somebody has a positive calcium scan or they have a family member die and they
think you know what I need to go get checked out they go to the doc without
and say why don’t we do a stress test and the doc says that’s a good idea.
You do a stress and then it goes down a bad path. Usually
there are better ways to go and one of those ways has to do with first finding
out that you have inflammation. You’re starting to hear about that more and
more. Even the TIME magazine had a cover article inflammation about cash over a
year ago… two or three years ago. So it’s even hitting the Leighton professional
medical non-medical press but it hasn’t quite hit the medical community yet. It’s
clearly not the standards. I think it’s going to take another ten years or so
before the standards catch up to the fact that cardiovascular inflammation is
a major cause of the medium risk for heart attack and stroke. Now those of
those of us who are on the channel those of us who have been watching the channel
know that we released a video a few weeks ago talking about how you can
actually go to the lab these days and order request and order your own
cardiovascular inflammation panel. You have to do that sometimes because again
your standard docs are not quite up to speed on that issue. So if they’re not
quite up to speed on it and they’re supposed to be the professional then
quite often they they criticize it and don’t want to go there. Now after
unfortunately after releasing that video it was an old video by Jon Lorscheider I
heard it was accurate at the time but since then our major source for for
getting harder vascular inflammation panel is it has closed their shop for
patients to go direct. So again Bart I keep using your name
Bart Robinson by the way says good evening from
southern New Jersey and again correct me if I’m wrong but Bart and maybe some
other folks have said, look yeah we’ve got to do the inflammation testing, we
know for example some folks have done it. I talked to I think it was nutritive up
this past week who saw this on our channel and went to quests and got the
inflammation panel and he’s actually been following him himself in his own
condition for quite a few months on it. He acknowledged… he said, yeah they shut
it down. So here’s one of our program points. I’m still able to get it in fact
I did not know that the that they had shut it down for patient direct requests
so that was one of the things that I wanted to talk about tonight. We are
we’re working out a way where we can make the inflammation panel available to
folks through our contract with Quest Labs now. I had a… I had a full-blown
slide deck which gave you details on it both about cardiovascular inflammation
and about how to get that through our through our contract. Unfortunately
that’s what bleep up. Yeah. If you’re gonna show your screen in my face to or
even just your screen you’ve got to go through a custom YouTube live and we had
both Cliede and Michelle couldn’t get it done but we do have our pilot
available. Here’s the thing. I’ll give it to you verbally if you would like to to
access the inflammation panel. Pardon me I’m going to go the old shaky paper
low-tech route again because the tech failed us or we failed the tech. Michelle
I hope you’re okay. Well here we go. Yeah “[email protected]…” I don’t have
I don’t have a black pen even so here we go
“…” Again and be patient with us we’re going to get
the technology there soon and Michelle I’m going to give you or give them your
business number if you’d like to order your inflammation panel cardiovascular
and inflammation panel through us. Another way to reach us phone number
wise is give me just a second okay six one five eight five three eight eight
four three. Now somebody told me one time quit apologizing for your technology.
I’ll quit apologizing for our thing now when we get it right. Again my apologies
but you can call Michelle at this number six one five five three eight
eight four zero three. And Michelle can help you get set up on ordering your
information panel with that order. I forgot what price we’re setting it up
as I think it was like hand on now I think it’s like 250 total or 350 total
and what you give with that is the labs themselves plus three months access to
the webinar program where we will actually be talking about the results of
the cardiovascular inflammation panel. So you’ll get three months access to a
weekly discussion and review of lab tests and discussion about how to how to
interpret them and you will be recording them so you’ll get that access to the
recording for the three months as well So I’ll go back to a couple questions.
Jeff Earnshaw what is the best way to test for cardiovascular information. If
you guys will bear with me I’ll go through some some of this verbally we
use for tests you could use other tests as well but let me just go verbally
through the process and then the four tests on the panel
you know. 147DegreesWest I see you’re here [email protected] Back to the point 147DegreesWest brought up the point of our bank that we were not talking that much
about the glycocalyx. The glycocalyx is a fuzzy lining of the arteries. What we are
finding is in the science is becoming very clear. We used to think and most
docs still look just at LDL. Well here’s the problem of looking at LDL has. The
people that have a heart attack have a normal LDL so sooner or later we’re
going to figure out that it may be it’s maybe not just LDL and in fact there’s
plenty of evidence it’s becoming clearer and clearer that it’s injury to the
intima and that intima is that fuzzy has the glycocalyx which is the fuzzy lining agenda that’d be the wrong number I don’t think so so once you get the
injury to that that glycocalyx. Then no matter what your LDL level is the the
LDL can get through that glycocalyx. That’s when the trouble starts. That’s
when you start developing plaque because plaque is nothing but LDL that has
become oxidized and lodged underneath that intima layer. So back to your
question Jeff Earnshaw what’s the best test again. We use a panel. It’s a panel
that was available direct to patients where you could order it yourself. Again
we found out the hard way a few weeks ago that it’s not available direct to
patients so we’re going to be making the right through our contract with
Quest and Cleveland Heart Labs. We look at my lab… peroxidase, LP-PLA2, HS-
CRP (that’s C-reactive protein). LP-PLA2 is PLAC2, that’s another
term we use for it in pl hold on just a second pardon the tech issue but we’re
getting yes blood yes yes I’m live now yes sorry
okay so again PLAC2, MPO, HS-CRP, and the most important one is actually a
functional test to look at whether or not the intima layer is intact
that’s called microalbumin-creatinine ratio and actually that’s a test that
are that’s done quite often by doctors in standard curative medicine. They look
at a different a different cut point and they’re looking at a different thing.
They’re looking at the overall function of the kidney. The kidney is nothing but
of each kidney is a million filters and each of those filters is called the
glomerulus. You don’t need to worry about what’s called and a filter is nothing
but a membrane supported by a structure. Here’s why that’s important.
The membrane in the kidney filter is the intima. It
is that lining of the artery wall so if that intima is injured it will let
protein out through the urine. That protein is the most common protein in
the blood and that’s called albumin so if you have a microalbumin spilling in
your through the filter of your kidney, you’re having microproteinuria and that
shows you’ve got damage to that intima. And guess what that has been shown to
microalbuminuria or leaks of albumin through the kidney filter has been shown
to be a significant risk for heart attack and stroke. So those are the
things that we use again. Pardon the interruption is because the
interruptions I’m just going to repeat a real quick: MPO, PLAC2, HS-CRP, and
microalbumin-creatinine ratio. And those are the tests that we’ll be making available
through through our contract with Quest. Again I talked to… I’ve talked to several
viewers who are getting it on their own and again it got cut off so we’re gonna
make it there. We’ll also make it available with access to webinars which
help us as a group as a community interpret these lands. So thanks for your
patience on that explanation. I’m going to go back and and go through some
questions and it’s working now. Slightly out of focus. I’ll tell you what slightly
out of focus is one of the least a month our tech problems right now but thank
you for letting me know the bit rates slow here. Let me go back and explain
what as you’re seeing. I’m not in my usual million dollar la studio yeah is
you know I’m being sarcastic. I’m not in my usual studio. I stand up there. I’m
actually in the you can see I’m in the offices here at K Health. Some of you may
remember it. I’ve done a couple of videos about K Health.
K health is… I drew an analogy between K in medicine and deep blue and chess.
Remember people say people used to think that computer logic would never be able
to beat a human in some deep relational type thinking like chess will be here. if
you listen to Garry Kasparov, he’s the guy that was the reigning champion that
got beat by a computer and he said I’ve been at once then I lost and I’ll never
get will never go back. Computers are better now at chess than humans. We had
never until now truly applied the power of computer logic to critical things
like making a diagnosis. K health is doing that and I’m a consultant K
Health. I’ve been working with them for a few months actually they just came out I
think I haven’t seen it I heard about it there was a there was a Forbes magazine
article recently about K Health being one of the new startups in the health
space to keep an eye on K Health. You can go to the app if you have an iPhone
or most of the other phones going and download K Health okay like the
letter K and health and then you can start asking it questions. You download
your own demographics to start asking questions and then it starts giving you
some potential diagnostics a diagnostic situations it’ll go through this ain’t
the usual disclaimers and here’s the thing. K Health has about five million
doctor-patient interactions already programmed into it so it’s looking at
experience and there’s no doubt even somebody as old as me that has five
million patient experiences to to draw so you start getting some advantage
there. Here’s another advantage that
the computer brings to the table and that is you know a doc has think things
like frustration technology people waiting to go out to dinner your family
problems whatever. Computers don’t have that problem. They think and go back
through everyone at the logic points when you bring that to the computer. Now
if you’re thinking or thinks that computers are going to replace doctors,
no, they’re not. Doctors will always have a critical place and still do always
will the doctor needs to communicate with with the patient. The doctor is the
one prescribing. So what K Health had and what I originally set up for K
Health was a doctor piece so now you go through the K Health process. They’re up
to 37 states now the thing with access and at the end of going through that
that diagnostic exercises with thee with the app it’ll say, would you like to
speak to a doc. I’ve been one of the doc except on the other end. When they should
said yes I’d like to speak to a doc and we and there’s some text and telephone
type of interaction. What’s exciting and what we’ve been talking about meaning
about today is setting up chronic care what it’s been in the past and so far is
more of an urgent care type of interaction like a digital urgent care
access. We’re having some exciting discussions about providing not only
digital urgent care but helping you and improve your access to high-quality
chronic disease care. So very obviously that it’s not ready… not ready for
primetime but it’s something that is very very exciting for improving cost
efficient much better quality and much better access to care in this space and
the things that we talk about K. Is coronary information caused
primarily by carbohydrates? Radnally. I would tell you you’re gonna get a whole
lot of people there’s a boatload of debate about that whether it’s carbs or
plant-based versus animal-based. I used to be plant based in my focus and
approach actually for almost 30 years. I will tell you though the research is
coming out now. It’s becoming clearer and clearer the original thoughts about
plant-based were then. LDL is what makes that most of the plaque I’d love to be
able to show you some of the research and some of the images that we have on
the deck but again no no images tonight. The bottom line is the research is
becoming clearer and clearer. It’s not the LDL. It’s not the animal products so
much. It’s the carbs. They’ve shown that four or five hours at 180 especially at
200 and it’s like a lawn mower came through and just cut out 2/3 of your
glycocalyx on the intima of their arteries injuring that intima and making
it very possible for LDL no matter what level you had to go through they dug
located so I think it’s becoming clearer and clearer that it’s the carbs are a
major major issue now. If you’re a 20-year-old or if you’re a 40-year-old that
doesn’t have any insulin resistance and your body is keeping your blood sugar
down 100 or 90 or 80 no matter what you eat, then no carbs are not causing a
problem. That’s one of my pet peeves with this whole debate of plant-based versus
animal-based or whatever else. There’s this assumption that there is the same
diet required by everybody. And I can tell you I’ve got a mother-in-law who’s
95. She’s got a lot of in stores this is
pure brown blonde diabetes. I haven’t insulin resistance. I’ve met one of the
criteria for diabetes at one point. I did have a blood sugar go over 200. I don’t
take insulin and most of us will not ever will not need to. I don’t
think I’ll ever need to. My son, on the other hand, is 24 years old and I
don’t think his blood sugar’s ever gone over 110 so he can eat all the carbs he
wants. I cannot enjoy my mother-in-law’s certainly cannot although she does she
doesn’t listen to me about the carbs okay something that alone for a while
Trump Tribe 2020 hello from Las Vegas well thank you very much for identifying
where you’re coming from and hello to you
Cynthia Armstrong hello from Chicago hello Cynthia
Brad Jackson what are the type things because inflammation and what are the
two or three best dietary foods or supplements one can take. Okay so the top
things that cause inflammation is I said a few minutes ago by far the most
prevalent problem is insulin resistance or full-blown diabetes. Insulin
resistance, metabolic syndrome, pre-diabetes… all you can get into some
academic arguments regarding the potential differences between those
three things. They’re all basically (for practical purposes… our purpose) is the
same thing. And guess what? They’re really the same thing as diabetes as well the
only difference is it’s a matter of degree. So if I’m routinely at a position
where if I eat a few carbs and my blood sugar goes up to 180 then number one I
need to avoid carbs. That’s one of the most important things I can do if my
assistance were to get worse you know my blood sugar to routinely go over 200
then I’d have full-blown diabetes. So that’s the only difference between
pre-diabetes and diabetes. As part of that conversation you can see one of the
major and most prevalent causes is pre-diabetes for causing inflammation. Why is that most? what I mean by
prevalent comment I’m sorry just the most common cause why is that common
there’s there are three major reasons why pre-diabetes is so common. One is the
aging population. As we hit our 40s and 50s especially as a better 60s by our
60s over half of us are pre-diabetic. The University of California in LA the
School of Public Health did a survey in California two years ago and they saw
over one-half of people adults in California time
at PETA use or does now you would say okay that’s sixty-year-olds.
So this is an issue of aging. They started at age thirty so there’s some
differences in terms of demographics, race, and clearly age. But pre-diabetes is
very very common now. Here’s the other thing that CDC is very conservative but
even the CDC will say there’s at least nine people out here in the world that
have Americans that have pre-diabetes that don’t know it but for everyone
nine or ten people have it and don’t know it for everyone that does know it
they’ll also say there’s for those of you who are not medication haters and I
know that’s there’s a lot of medication haters there’s not a lot of non-haters
point about metformin for about every person that’s on metformin there’s
probably at least thirteen or fourteen that need to be on it.
Now again half of my patients don’t want to take any kind of medication. I
understand that. I actually delayed my own medication for about three years for
similar reasons. Now you said what are some great
supplements. Here’s the thing. Here’s the bottom line comment about supplements
and lifestyle. Number one, by far the most important thing in terms of managing
free diabetes is lifestyle. You cannot change younger although (I can
see that I am having some connection problems) you can’t change your age in
terms of getting younger but you can improve dramatically. One of the most
common things I see is that people have a BMI there are up around thirty meaning
they are too heavy and they have increased fat mass that is incredibly
important in terms of seeing patients dealing with folks that
is the most common thing then you have. Some other things as well.
Inflammatory diseases like rheumatoid arthritis, lupus, inflammatory dermatitis.
Those things can cause inflammation as much as insulin resistance or
pre-diabetes and then we talked about one thing we didn’t talk about I haven’t
mentioned it because so few of my patients and subscribers are Swiss is
nuclear to your arteries in terms of inflammation so it’s not just smoking
it’s tobacco products and it’s also vaping so I’ll get off of that topic for
a while. Doc you’re right I am leaning from this chat I think he meant not
learning hundred knows Charles Ransom Thank You Charles
and then 47 degrees with [email protected] prevmed, it’s [email protected] OMG
old Alabama Gardner if you guys haven’t seen Oh a juice channel you should take
a look he is the original and best farm-to-table he’s far better organized
than I am he actually used to manage do maintenance on rockets so and he’s down
in the Alabama areas avid farmer and actually takes you in his videos through
how to plan a garden how to clean out the garden headed home prepare the soil
Adam get the right all of these things he takes you through about three to four
months worth of development and growth and then at the end he’s got this
plastic thing he eats what he’s making whether it’s a vegetables of whatever he
says mm-hmm I wish I could share that and I wish it could anyway old Alabama
gardener so Dr. brewer how does homocysteine plasma test play
in hardened artery health that’s a great question I’m assisting if you get levels
up around 17 and 18 has been noted is a heart attack risk that actually
gets into to go things like Micah leptin a whole lot of people love to hate on
Stephen Gundry and I get irritated this well because I mean he’s got some really
good stuff what’s proven is here but then there’s a whole lot of other stuff
sometimes you have to get through and that can be frustrating they tend to
think the same thing of the issue of methylation but I’m gonna go there with
methylation this homocysteine issue that you brought a AG does have to do with
methylation and I know I’ll get a lot of haters about this issue but if you look
at the science there is real and valid issues and scientific evidence
supporting the issues around methylation unfortunately just like the lectins
issue you get a lot of theory and unproven John but you there’s a lot of
facts inside some of the facts around homocysteine and methylation are I’ll
just methylation is a very very big issue and when you have a very high
homocysteine level what happens is the the metabolic processes excuse me are
getting backed up and you’re getting a buildup of homocysteine because it’s one
of the metabolic feeders into a broken methylation system so if the methylation
system is broken you get you get buildup of homocysteine there’s some
practical things you do most practical easel thing is to take methylated
b-vitamins one of the more common ones is thorn and I’ve taken thorn methyl
guard and then you’ve got thorn phor in the thorn metal guard plus and I’ve
taken that for years because I’ve got a methylation problem in terms of
methylation and again you’ll hear a lot of docs say oh they looked at it it’s
not real they haven’t really looked at all of the science as many of you know
I’ve worked I did some training in genetics and at Stanford and did didn’t
have worked with a genetics lab my genetics for about four years now
so I’ve got easy access to methylation testing and I have like a third to to a
half of our population I have over I have about fifty percent decrease in my
methylation function when I study a patient with an elevated and methylation
elevated homocysteine how do you tell them we could get methylation genetics
but on a practical basis why don’t you just take some some methylated B
vitamins I actually am coming up and I’m getting
a potential access to a source for that and may may start offering that through
our channel as well for those of you who have interests in this area I would just
tell you I did a couple of videos a couple of years ago on the concept of
what’s called a Reds a or EBS it’s a it’s a place where they found
that you actually get decrease in blindness associated with some very high
levels mostly of antioxidants there’s some methylation focus in and is again research which is kind of
great institutions like the the ophthalmology program in mind I’ll just
leave that topic alone thank you for bringing it up Oh AG Cynthia mom Cynthia
Armstrong would you please do another video on what to eat your information
keeps improving and advancing and I would really appreciate well thank you
very much Cynthia I would love to I won’t promise that I won’t get it out I
mean then I’ll get it out any time over the next two weeks because as you can
see I forgot we have a lot of neat stuff that we’re working on and I have to tell
you I need to I need to do some updates to a lot of mom my most recent what I
eat videos are probably a year or two old and I’ve actually made some more
changes I did some more research on the issue of TMAO and I won’t go there
tonight but if you’d like I’d like to I’d like to do that Bjorn to fly thank
you for attending you had a video about why most research is wrong with John I’m
gonna butcher the name you need us it’s a Greek name by the way
Johnny and ADA C’s at Stanford how do you cook with his findings and
especially research and diet almost everything is wrong and a dice would be
just as good hmm that is a key issue especially in
prevention because we know the top three priorities in terms of lifestyle are
diet diet and diet and then sleep and exercise and you know several other
things go on so you know I’m in sort of a no-man’s land in terms of science and
what I do for a living for this those of you who don’t know I got my start in the
ER and got focused on prevention because most things that take people into
or like heart attack stroke even a fishhook in the nose or thumb are things
that could have should have been prevented so I went to the best place
that at the time may be still the best place available for prevention training
was John Hopkins and loved it ended up running the program there and have been
doing prevention ever since now the reason I bring that up is my original
training there and my function was in research so I would clearly agree with
Anita support that diet research is a bad is a top place you know there was a
lot of research that came out just over the past six months regarding carps and
there’s still a lot of stuff there’s just not known I mean he you can see me
the nonverbals are true it’s like sometimes you just want to scream but
there’s some things that then you know things that you see things that you work
with on a day to day basis I don’t care if you’re a plant-based guy I don’t care
if you’re a babe paleo fan I don’t care if you’re a
low-carb keto carnivore I don’t know anybody that’s gonna argue
that having a BMI of 30 is healthy unless you are built like Arnold
Schwarzenegger at the time that he won Mr. Olympia I know there’s no no I don’t
know Flores Negras politics or any of that in here but here’s the point a lot
of people especially men think I get this all the time oh I’m big-boned
there’s a lot of muscles aren’t words and Eggers BMI at the time that he won
Mr. Olympia was 30 so we’re not talking the research reason the research is
clear and clear that it’s not so much having weight it’s the bat mass and if
you look at a lot of the research around diabetes
cardiovascular inflammation and fat you see that we used to think that was just
storage it’s not fat is a an endocrine and endocrine organ and it creates some
adipic kinds and some other things that create inflammation so the more the
bigger your fat mass the more inflammation you’re driving it also
drives insulin resistance so you can say well that’s not nutrition the bottom
line is one of the major drivers in the nutrition space we already know and that
is BMI or fat mass so you can push that aside and say it doesn’t matter which
Dietary place you come from we know that one of the key points is how much you
are storing in your fat then you can start taking some other logic issues as
well yes I’m very much aware that there’s science and good science that
came out recently about a J curve regarding carbs I don’t think I think
the jury’s still out on a lot of that stuff but I think there’s also some real
clarity as well I think there’s plenty of information that would indicate that
you don’t have to get into the what you need assists of–can about in terms of
dietary research we wouldn’t even call it dietary with certain research that
shows if you’re diabetic if you cannot metabolize carbs and you
eat carbs and your blood sugar goes up and it stays over 200 810 24 hours a day
then you’re burning your arteries you’re creating you’re gonna blind your
arteries and your eyes are gonna I’m gonna get inflamed and burned out you’re
gonna get blind you’re gonna get stroke you’re gonna get heart attack it’s said
I mean all of these come from the same place so
yes dietary science is it can be a quagmire but here’s the other thing
about that even within the dietary science it’s like over half of all
science is wrong I won’t argue that and yet again one of my major things on on
you YouTube is to try to show what is the scientific evidence what’s and
here’s one of the questions what is good science and what is not so again we’ve
gone on quite a bit about that I think it’s a great question I hope this was
helpful Jeffrey Scott Fisherman thank you for joining on kita feel great all
aches and pains in joints gone lost fifty pounds five foot six weight 143
pounds congratulations Jeffrey Scott Fishman you keep that weight off and you
may you clearly from a heart attack risk perspective and dementia and all these
things that just went through blindness all these other things you may have
saved yourself a decade I personally think more than that maybe two maybe
three thirty one inch waist excellent blood sugar 85 in blood pressure 102
over 65 triglycerides were 72 HDL is 60 X LDL is 160 now a lot of people would
say oh that LDL is terrible you need to go on statins you need the panic and the
concern I’m actually you know you this brings up a good point and I’ll just
make this point I had it’s like three people just over the past week get
started with me and or call me to get started because they went on kita they
lost weight and their LDL went up and there doc got panicked and there is a
thing out there called low fat
hyper is LDL responders bottom line what they’re
talking about and I think it’s Dave Feldman he’s got a whole bunch of
channel on that he’s one of these engineers like Ivor Cummins got some
very good logics got some very good experience some very good data if you
look this up you had a question about a diet I first got a good a good section
on it as well but a mine is a lot of people maybe you 20 percent of folks
will lose a good will be in even though they lose a good bit of weight on keto
diet will get a significant increase in LDL they will panic or their doctors
will panic and then sometimes they will panic and here’s what I would say you
know what I if if I would much rather have that 40 what 50 pounds gone and
deal with the LDL of 160 and it’s really debatable right now excuse me it’s
really debatable right now how much risk if any that LDL of 160 is creating
there’s no question in my mind that it’s a lot less risk than the 40 to 50 pounds
for creating before you lost them so I’ve got a lot of patients that are the
LDL hyper responders or lean mass hyper responders is I think the term that
David Feldman uses getting more and more of those coming in to me it’s it’s a
great world to be in it’s a it’s a great thing over half of my patients now by
the way or people that are coming in saying I’ve lost over 30 pounds and my
response is what it was to you Jeffrey it’s like well you’ve done the major
heavy lifting the hard part what can I do for you why are you here to see me
and almost all of them say the same thing that’s right I know I have done
the hard part I know that I’ve made a major change to make but I still want to
dial it in and there’s no question in other words continue to improve my life
I still improve my risk situation and there’s no question in my mind my
response is you can always improve your risk situations so it’s a very it’s a
very nice place to be gibberish catfisherman should I worry
over LDL I think we just went over that taylor white thoughts on sedimentation
rate is a marker at all for cv inflammation you know that’s a really
good question I don’t know the science this the evidence around that one of the
things you have to wonder is how much of that is the case but let me just add the
risk of making a fool of myself and as you guys know I mean the start of the
event was yet another more evidence that I’m not afraid to make a fool of myself
so from from what a little I know about the sed rates it is a little bit more to
do with the some of the proteins in the in the body and quite so much to do with
inflammation then actually brings out the actual question what is inflammation
you know you hear about inflammation you hear about information and result in
relation to cardiovascular risk unless you know specifically what it is it can
be kind of confusing here’s what it is it is taking friendly fire from your own
immune system so what happens is your immune system recognizes that this LDL
is not supposed to be in your intima media space and what and what it does is
it when it is the same thing and it does any kind of thing that it recognizes
that shouldn’t be there whether it’s a tumor a tumor cell or an infection or
foreign body it starts sending in antibodies it starts sending in things
like immune cells T cells monocytes start coming in they release enzymes to
digest that LDL and they also release things called what we call chemo tactics
side o tactics they you know big technical medical rings but in other
words they’re sending out an alarm to other
sells that there’s a problem here there’s foreign body we want to clear it
out here’s and all of that’s working fine up until this point it actually
does its job it actually dissolves that hard waxy
plaque in the artery wall unfortunately those cytokines those immune alarms
those chemicals those enzymes if they break back through the intima layer into
liquid the liquid flow of the bloodstream that can cause a clot if the
clot is big enough it goes to the heart and it goes to the heart that’s a heart
attack if the clot is big enough that it goes to the brain that’s a stroke if you
know you have situations where it’s microscopic on there wiping out a few
hundred if you’ve got a few thousand cells at the time but that’s one of the
major mechanisms for dimension blindness kidney disease are heart failure because
if you look microscopically at the tissue you get three decades and
continuing to have these microscopic clients even though they’re only wiping
out a few hundred cells pretty soon 24 hours a day for a decade that starts
chewing up the tissue whether it’s brain tissue art to stimulate Jack Frost’s can
you repair the gym called not sure at all what that makes ran out and thank
you at sixty in it the CAC kept coronary house counting sports 673 I’m trying to
stay low carb I’m low carb man I’m not like rabid keto and I’m certainly not
carnivore I may try that at some point but I think that’s wise from unless you
know exactly what your status is how well you metabolize parts once you get
sixty especially 65 and 70 shows sooner and some system until proven otherwise
by the way we are we’ll be offering those and intense
survey and these are three quests so again that’s something if you’re
interested in finding out to me I would go with the insolent survey first and
just see what sort of problem I had actually I would probably order both but
again there’s some conversation you can have with Michelle you can look through
some of our videos more understanding if we’re wearing or what your wrist might
be and you know what your interests are what you thought David Jones I think you
might be missing the damage from hyperinsulinemia David thank you very
much I was and I tend to leave that out so here’s the here’s the thing so David
thank you so much for bringing that up I will tell you it was about we’ve been
trying to get Quest in CHL to add insulin values to the OGTT values for a
year they finally agreed about three or four months ago so now in addition to
getting just OGTT an oral glucose tolerance test where you take it fasting
glucose take the 75 grams of sugar fructose and then one hour later you
measure the glucose again one hour later you measure the glucose zero one in two
hours probably two maybe three times a month here’s what I’m seeing David I
remember a patient what last week or the week before their initial number was in
the 80s that fasting glucose was in the 80s there one hour went up to like 115
their two-hour glucose was back around eight nine most people will say that’s
those are really good numbers but once we were able to get insulin there the
initial insulin was what 10 15 which is too high for a basal insulin in order to
keep down to a little bit below 120 that his
body was using over a hundred of insulin had to use over a hundred of insulin to
keep that one-hour OGTT down below 120 and then it still
was up in the high 30s at two hours so again to your point David Jones that
individual even though their glucose numbers are great
they’ve got significant in flim and flam agem pardon the technical term it’s
they’ve got significant drivers for inflammation and it’s not coming from
book oats it’s coming from insulin insulin does I mean there’s clear plenty
of clear evidence heart attack risk in people like I just described is one of
the key areas where you see that evidence that insulin drives
inflammation as well thank you very much David for that reminder
Jim Meili hello from Thunder Bay Canada hello thank you very much for for
letting us know where you are you know what this question thing just flipped on
me I’m gonna miss a few Rock of to7 hello doc Peter Atia has said he doesn’t
care if your plant-based paleo keto of whatever as long as your blood Sugar’s
stayed at a good level with low variation I would agree with Pete him
Peter he and I share a little bit of stuff I started off as an ER doc he
started off I think as a surgeon and we both worked at Hopkins ve Russell I
think I have more hair than Peter and I think I’m a lot over but for the most
part I I agree with that I agree with you I appreciate you bringing it up
anybody with any sense is gonna say you need to manage you need to eat to the
glucometer in other words know what causes your
blood sugar to go up and if rice makes it go up be careful about eating rice I
will share with you most probably Minnesota hits
videos if I go on the proline diet the you know the probe doesn’t even know the
proline that is fault or Longos diet it’s there’s a little bit more of a plan
based approach to it because he wants to decrease fat calories and protein
calories at least for that five days and he doesn’t good research showing that it
helps with insulin resistance but here’s the thing to remember and here’s one
thing that showed I had the my Libra it’s a continuous glucose monitor on and
about 20 minutes after I eat the suit there’s two soups in there you go to
endo about 700 calories per day for about five days your you do have two
meals each of them is the like a powdered soup on those pattern soups
that will take my blood sugar up to about 180 now than they yes
180 so the next thing that you might ask would be well Brewer how long does it
stay there it’s critical how long it stays there and I’ll tell you I don’t
know because I’m watching it on my my CG Empire glucometer continuous glucometer
and I know that if I started to get a spike like that I can go out and take a
walk 20-30 minutes and you it’ll usually take the top right off of that but again
that makes the point eat to the glucometer in the point that you brought
the quote from Peter Atia doesn’t matter what your diet is it matters what the
impact is on your blood sugar and I would add as David pointed out it also
matters the impact on your insulin I’m gonna butcher this name I’m sorry Liao
findi hi doc a question do you know if row row ghost and I think you mean
rosuvastatin I hope that’s what you mean omega-3 cookin the girl any platelets
because side-effects of bruising echo hidden grout clearly your copa de graal
is an antiplatelet drug as you mentioned and yes any of the antiplatelet drugs
can cause side effects of bruising the red and Dylan ham over attracted
Richard Marx hi Dr. Brewer how does one improve one’s HDL level through diet FYI
I’m from the UK thank you for thank you guys for letting us know where you’re
from one of the first things I’ll do when I see a patient even if I don’t
have the insulin resistance almost everybody is going to come to me
with a with a cholesterol panel and I can get a really good handle of their
risk very quickly and I’m not looking at LDL I’m looking at triglycerides and HDL
and here’s what classically happens if you get a triglyceride over HDL that’s
elevated it indicates you probably have insulin resistance and or diabetes
in other words too many too many carbs now how does that happen well insulin
has a couple of effects not only does it it wants to get insulin is made and to
effects that it has both result in getting dropping the blood sugar the
first effect is to move blood I mean blue sugar out of the blood and into the
cells where it’s safe the second and a lot of people don’t
know about this is insulin shuts down fat burning well fat burning is
typically usually triglycerides that’s what your you know your love handles
your subcutaneous fat is so if you are constantly in a place where your muscle
and liver tissue push back and resist insulin excuse me your punts constantly
in a place where your blood Sugar’s hanging around 160 180 – high insulin
bet and used by the wave in are hanging around 30 when they need be less than 5
on a basal level not on a fasting level up around 30 then your insulin is
obviously not being successful at getting usually not getting getting the
sugar from the blood in the cell in addition though it’s shutting down fat
burn so that’s why your triglycerides are going way
why did I go down that bunny trail because of your question of how to
decrease or increase HDL cut the carbs increasing the triglycerides increase
having a high continuing insulin level high triglyceride level tends to chew up
HDL I won’t go into the technical reasons why that’s the case but cutting
carbs is a great way to increase HDL another way to increase HDL is to excuse
me there is a supplement niacin I know there’s a bunch of niacin haters and
probably in any any audience there were a couple studies ain’t high and I can’t
remember the name of the other one that talked about oh nice and doesn’t work
that’s not true I’ve got three or four videos on niacin and niacin does tend
to help I’m gonna we’re getting long in the tooth had a lot of great
participation I would ask you I would s you again if you would help us buying
and helping us with our pilot on offering the inflammation panel I forgot
to put in a plug for some of the other activities we’ve got going on I’ve got a
book coming out in not sure it when hopefully this fall it’s all about how
to detect plaque we’ve got an event a full event where you can come in get
your CIMT get all your labs and talk about those with a 2-day boot camp type
of environment then intense focus on your lab results and how to interpret them
what to do and what some of the risks are the science that we’ve been talking
about tonight for about an hour that’s in Louisville you can find that on our
our website if you don’t want to do the labs you can come in and not do the labs
just get the see I am too many people have been asking how to get a CIMT
we’ve set up IMT access tour intima-media thickness
test it’s a plenum it was an ultrasound that
we take up the carotid artery and shows how much plaque you have in there ninety
nine or nine hundred and nine ninety-nine out of a hundred of these
ultrasounds are not at ninety most of them just look to see if you have enough
plaque to impact your flow this is a different type of thing and uses
software to detect it more than if you have an impact on your flow how much
like you actually have because the vast majority of strokes and heart attacks
occur in people that don’t have enough plaque to implant the flow and then
stress test is a flow study but just a lesser amount of plaque so that’s why
you look at to see just how much black we have you can do that with calcium
score the advantage to calcium score is that it looks at the heart
the disadvantage to calcium score is that it only looks at counting it
doesn’t look at that soft plaque that creates the immediate danger that we
talked about early on we do have a couple of courses that we we just
finished a third course we’re getting that up and rolling we’ve got a course
on cardiovascular inflammation what it is how to understand it and again it
goes into a little bit about how to get some of the testing yourself that’s on
the membership page if you haven’t heard about the membership page that’s on the
website and the website is thank you very much for your interest and I’m gonna
sign now well I think I mean you


  1. Thanks Dr I just retired from being an RN for over 40 years of patient care. I use to be a Med Tech and worked in the Lab analyzing blood for 7 years prior to Nursing. I am now 71 and from one year that is last year to this year, I developed from a 10% right side carotid plaque to 35% this year and want to try and reverse this process so I can be happy and active until the end. Also from Las Vegas Nevada.

  2. I have been told that when you fast for the Cholesterol testing, this causes the liver to produce more LDL especially when you fast for 12 hours

  3. I just did some searching and some math, I think there is a way to get the following labs direct for a little over $260 (+ tax I assume):
    -MPO (myeloperoxidase)
    -hs-crp (c reactive protein)
    -24 hour microalbumin creatinine urine sample
    -lipid panel
    -3 specimen oral glucose tolerance test (2h).

    If you'd like some more info, I can try to write up the details

  4. Dr. Recently my son in his 20’s was put on a statin and blood pressure med (he has white coat syndrome so we don’t really know if he actually has high bp) because of Familial Hyperlipidemia and his Dad has just had a OH/triple CABG and has also been found to have moderate Aortic Stenosis, which they said was genetic. I FH but the last three years I’ve been eating clean, IF and OMAD most days, have lost 100 lbs but 150 to go- I have low triglycerides, good HDL and 218 LDL. My son finally quit sugar and started eating very healthy after his Dad’s emergency surgery, thankfully he is very fit and always had been in the 50% weight percentile growing up.
    After his surgery, My husband was put on two bp meds and Atorvastin. His cardio doctor insist he take these for life and wants my son on them for life, as well. My husband had no idea he had three blocked arteries and has never been ill in 57 years except last year has had low testosterone and a little high PSA until he was sick with pneumonia recently and felt he wasn’t getting better so he went to the ER and was in emergency surgery within the day.
    Im worried about the bp meds and statins for life for my son, especially. He is so young. The docs all said because of my husband’s surgery it is a given he has to take these. My Husband is also on Warfarin for at least a few more months.
    Would you agree with this for both of them? Is this updated advice?

  5. You are seeing part of the story on BG and insulin levels that Dr Kraft lays out in his book. The 4 hour story with 15,000 data sets is very important and illuminating! It's a short but valuable read…

  6. Dr Brewer. I’m 56 year old woman. My CAC score is 138, family history of heart disease . LDL 160, triglycerides 111 and good blood pressure and good A1C. My doctor put me on 20 mg Lipitor, I asked for low dose. I also have to do a stress test next week. I ordered my own CRP which came back at 6.48, very high. I’m on a low carb, no grain, so sugar diet. Is Lipitor and diet and exercise my only options?

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